alzheimer's disease

The current medical thinking is that Alzheimer’s disease (AD) is a deficit in neurotransmitter function; specifically acetylcholine. Doctors have become obsessed with the characteristic amyloid plaques which are found in Alzheimer’s brains. But there is no evidence whatever that these plaques are the cause of Alzheimer’s. In fact scientific studies say the opposite. Drugs designed to reduce the presence of plaques have no significant effect on the progression of AD. Moreover drugs designed to rectify the acetylcholine deficiency also fail miserably, even though they are pushed with phoney science and TV advertizing hype (Aricept etc. are worthless). All this is despite the fact that there is a better model for AD. It’s better because rectifying it results in dramatic improvement. Alzheimer’s patients suffer from a metabolic disruption that inhibits the brain’s ability to use glucose to power neurons.

The current medical thinking is that Alzheimer’s disease (AD) is a deficit in neurotransmitter function; specifically acetylcholine. Doctors have become obsessed with the characteristic amyloid plaques which are found in Alzheimer’s brains. But there is no evidence whatever that these plaques are the cause of Alzheimer’s. In fact scientific studies say the opposite. Drugs designed to reduce the presence of plaques have no significant effect on the progression of AD. Moreover drugs designed to rectify the acetylcholine deficiency also fail miserably, even though they are pushed with phoney science and TV advertizing hype (Aricept etc. are worthless). All this is despite the fact that there is a better model for AD. It’s better because rectifying it results in dramatic improvement. Alzheimer’s patients suffer from a metabolic disruption that inhibits the brain’s ability to use glucose to power neurons.